VITAMIN D DEFICIENCY IN HIGH-RISK PRE-ECLAMPSIA PATIENTS AND LABORATORY BLOOD CHANGES

Хамраев Хумоюн Хамзаевич

Journal of reproductive health and uro-nephrology research (Репродуктив саломатлик ва уро-нефрологик тадқиқотлар журнали) · 2021-yil

Annotatsiya

PE is responsible for almost 100 000 maternal deaths annually worldwide. Despite some advances in the study of PE, it is still a major cause of maternal mortality with no clear trend towards improvement [1, 2, 5]. Recent studies in different countries have drawn attention to the potential of vitamin D to reduce the risk of PE. It is believed that the active form of vitamin D-1,25 (OH) 2D regulates the transcription and function of genes related to trophoblast invasion, normal implantation and angiogenesis. Vitamin D (1α,25-dihydroxyvitamin D3) exerts its effects on cells through both genetically mediated and non-genetic pathways. They include both long-term modulation of gene expression (genetic mechanism) and short-term activation of intracellular communication (non-genetic mechanism).

Maqola ma’lumotlari
MualliflarХамраев Хумоюн Хамзаевич
JurnalJournal of reproductive health and uro-nephrology research (Репродуктив саломатлик ва уро-нефрологик тадқиқотлар журнали)
Nashr sanasi2021-06-01
Jild1
Son3
TilRus

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Kalit so‘zlar

Преэклампсия, дефицит витамина D, беременность, репродуктивные и перинатальные потери, Preeclampsia, vitamin D deficiency, pregnancy, reproductive and perinatal losses, Preeklampsiya, D vitamini etishmovchiligi, homiladorlik, reproduktiv va perinatal yo'qotishlar

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